Like most companies in the smartphone game, HTC wants to pack its top-of-the-range devices with powerful camera tech, and that's no longer just a case of adding more megapixels. The new HTC One (M8), for instance, hosts a pair of cameras on its rear...
Notification Center collects all of the notifications your receive on your iPhone or iPad into one easy-to-access place. By default you can even pull down Notification Center from your Lock screen to see any messages or other notifications you may have recently missed. If, however, for privacy and security reasons, you prefer Notification Center not be accessible from the Lock screen, there's an easy way to disable it.
Now you should no longer be able to swipe down to access Notification Center on the Lock screen.
If you still want to see upcoming events for your day, you can always leave Today View turned on. If you don't want notifications available on the Lock screen, just turn off Notifications View.
Do you use any of these options to keep your notifications more private? If so let me know what options you have disabled and why!
For one day and one day only
You've probably heard that you'll find the cheapest airfare on a Tuesday. But according to a new study from travel startup Hopper, that's a myth. So when can you find the best fares?
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The latest version of the Google Translate app is making it even easier to have conversations between languages. The latest update, which will begin rolling out to users today, makes it easier to converse with someone by just activating the app then turning the phone between two people. You now simply select the languages you'd like to translate between, tap the microphone and speak your sentence or phrase, and turn it to the other person.
Gesture support detects when you turn to the other person, and translates what you said, then prompts them to speak back to you. Once you get going back and forth, the experience will be much more conversational than before, hopefully lowering the boundaries of speaking to someone in another language.
For times when you want to translate written input, the latest update also expands handwriting input to Hebrew, Greek, Javanese and Esperanto. If you don't want to write, you can always capture text with your phone's camera and translate any selected portion if you'd like — now supporting Malay and Ukranian.
Source: Google Translate Blog
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PUBLIC RELEASE DATE: 11-Nov-2013
ShareContact: Shawna Williams
shawna@jhmi.edu
410-955-8236
Johns Hopkins Medicine
A specialized type of brain cell that tamps down stem cell activity ironically, perhaps, encourages the survival of the stem cells' progeny, Johns Hopkins researchers report. Understanding how these new brain cells "decide" whether to live or die and how to behave is of special interest because changes in their activity are linked to neurodegenerative diseases such as Alzheimer's, mental illness and aging.
"We've identified a critical mechanism for keeping newborn neurons, or new brain cells, alive," says Hongjun Song, Ph.D., professor of neurology and director of Johns Hopkins Medicine's Institute for Cell Engineering's Stem Cell Program. "Not only can this help us understand the underlying causes of some diseases, it may also be a step toward overcoming barriers to therapeutic cell transplantation."
Working with a group led by Guo-li Ming, M.D., Ph.D., a professor of neurology in the Institute for Cell Engineering, and other collaborators, Song's research team first reported last year that brain cells known as parvalbumin-expressing interneurons instruct nearby stem cells not to divide by releasing a chemical signal called GABA.
In their new study, as reported Nov. 10 online in Nature Neuroscience, Song and Ming wanted to find out how GABA from surrounding neurons affects the newborn neurons that stem cells produce. Many of these newborn neurons naturally die soon after their "birth," Song says; if they do survive, the new cells migrate to a permanent home in the brain and forge connections called synapses with other cells.
To learn whether GABA is a factor in the newborn neurons' survival and behavior, the research team tagged newborn neurons from mouse brains with a fluorescent protein, then watched their response to GABA. "We didn't expect these immature neurons to form synapses, so we were surprised to see that they had built synapses from surrounding interneurons and that GABA was getting to them that way," Song says. In the earlier study, the team had found that GABA was getting to the synapse-less stem cells by a less direct route, drifting across the spaces between cells.
To confirm the finding, the team engineered the interneurons to be either stimulated or suppressed by light. When stimulated, the cells would indeed activate nearby newborn neurons, the researchers found. They next tried the light-stimulation trick in live mice, and found that when the specialized interneurons were stimulated and gave off more GABA, the mice's newborn neurons survived in greater numbers than otherwise. This was in contrast to the response of the stem cells, which go dormant when they detect GABA.
"This appears to be a very efficient system for tuning the brain's response to its environment," says Song. "When you have a high level of brain activity, you need more newborn neurons, and when you don't have high activity, you don't need newborn neurons, but you need to prepare yourself by keeping the stem cells active. It's all regulated by the same signal."
Song notes that parvalbumin-expressing interneurons have been found by others to behave abnormally in neurodegenerative diseases such as Alzheimer's and mental illnesses such as schizophrenia. "Now we want to see what the role of these interneurons is in the newborn neurons' next steps: migrating to the right place and integrating into the existing circuitry," he says. "That may be the key to their role in disease." The team is also interested in investigating whether the GABA mechanism can be used to help keep transplanted cells alive without affecting other brain processes as a side effect.
###
Link to the article: http://www.nature.com/neuro/journal/vaop/ncurrent/full/nn.3572.html
Other authors on the study were Juan Song, Jiaqi Sun, Zhexing Wen, Gerald J. Sun, Derek Hsu, Chun Zhong, Heydar Davoudi and Kimberly M. Christian of Johns Hopkins, and Jonathan Moss and Nicolas Toni of the University of Lausanne in Switzerland.
The study was funded by the National Institute of Neurological Disorders and Stroke (grant numbers NS047344 and NS048271), the National Institute of Environmental Health Sciences (grant number ES021957), the National Institute of Child Health and Human Development (grant number HD069184), the Dr. Miriam and Sheldon G. Adelson Medical Research Foundation, the Brain and Behavior Research Foundation, the Maryland Stem Cell Research Fund, the Swiss National Science Foundation (grant number PP00A-119026/1) and the Fondation Leenaards.
Related stories:
Brain's Stem Cells 'Eavesdrop' to Find Out When to Act http://www.hopkinsmedicine.org/news/media/releases/brains_stem_cells_eavesdrop_to_find_out_when_to_act
On Using Stem Cells in the Brain to Study Mental Disorders http://www.hopkinsmedicine.org/institute_cell_engineering/_includes/videos/Transcriptions/Song_txn.html
Hopkins Researchers Uncover Key to Antidepressant Response http://www.hopkinsmedicine.org/news/media/releases/hopkins_researchers_uncover_key_to_antidepressant_response
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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
PUBLIC RELEASE DATE: 11-Nov-2013
ShareContact: Shawna Williams
shawna@jhmi.edu
410-955-8236
Johns Hopkins Medicine
A specialized type of brain cell that tamps down stem cell activity ironically, perhaps, encourages the survival of the stem cells' progeny, Johns Hopkins researchers report. Understanding how these new brain cells "decide" whether to live or die and how to behave is of special interest because changes in their activity are linked to neurodegenerative diseases such as Alzheimer's, mental illness and aging.
"We've identified a critical mechanism for keeping newborn neurons, or new brain cells, alive," says Hongjun Song, Ph.D., professor of neurology and director of Johns Hopkins Medicine's Institute for Cell Engineering's Stem Cell Program. "Not only can this help us understand the underlying causes of some diseases, it may also be a step toward overcoming barriers to therapeutic cell transplantation."
Working with a group led by Guo-li Ming, M.D., Ph.D., a professor of neurology in the Institute for Cell Engineering, and other collaborators, Song's research team first reported last year that brain cells known as parvalbumin-expressing interneurons instruct nearby stem cells not to divide by releasing a chemical signal called GABA.
In their new study, as reported Nov. 10 online in Nature Neuroscience, Song and Ming wanted to find out how GABA from surrounding neurons affects the newborn neurons that stem cells produce. Many of these newborn neurons naturally die soon after their "birth," Song says; if they do survive, the new cells migrate to a permanent home in the brain and forge connections called synapses with other cells.
To learn whether GABA is a factor in the newborn neurons' survival and behavior, the research team tagged newborn neurons from mouse brains with a fluorescent protein, then watched their response to GABA. "We didn't expect these immature neurons to form synapses, so we were surprised to see that they had built synapses from surrounding interneurons and that GABA was getting to them that way," Song says. In the earlier study, the team had found that GABA was getting to the synapse-less stem cells by a less direct route, drifting across the spaces between cells.
To confirm the finding, the team engineered the interneurons to be either stimulated or suppressed by light. When stimulated, the cells would indeed activate nearby newborn neurons, the researchers found. They next tried the light-stimulation trick in live mice, and found that when the specialized interneurons were stimulated and gave off more GABA, the mice's newborn neurons survived in greater numbers than otherwise. This was in contrast to the response of the stem cells, which go dormant when they detect GABA.
"This appears to be a very efficient system for tuning the brain's response to its environment," says Song. "When you have a high level of brain activity, you need more newborn neurons, and when you don't have high activity, you don't need newborn neurons, but you need to prepare yourself by keeping the stem cells active. It's all regulated by the same signal."
Song notes that parvalbumin-expressing interneurons have been found by others to behave abnormally in neurodegenerative diseases such as Alzheimer's and mental illnesses such as schizophrenia. "Now we want to see what the role of these interneurons is in the newborn neurons' next steps: migrating to the right place and integrating into the existing circuitry," he says. "That may be the key to their role in disease." The team is also interested in investigating whether the GABA mechanism can be used to help keep transplanted cells alive without affecting other brain processes as a side effect.
###
Link to the article: http://www.nature.com/neuro/journal/vaop/ncurrent/full/nn.3572.html
Other authors on the study were Juan Song, Jiaqi Sun, Zhexing Wen, Gerald J. Sun, Derek Hsu, Chun Zhong, Heydar Davoudi and Kimberly M. Christian of Johns Hopkins, and Jonathan Moss and Nicolas Toni of the University of Lausanne in Switzerland.
The study was funded by the National Institute of Neurological Disorders and Stroke (grant numbers NS047344 and NS048271), the National Institute of Environmental Health Sciences (grant number ES021957), the National Institute of Child Health and Human Development (grant number HD069184), the Dr. Miriam and Sheldon G. Adelson Medical Research Foundation, the Brain and Behavior Research Foundation, the Maryland Stem Cell Research Fund, the Swiss National Science Foundation (grant number PP00A-119026/1) and the Fondation Leenaards.
Related stories:
Brain's Stem Cells 'Eavesdrop' to Find Out When to Act http://www.hopkinsmedicine.org/news/media/releases/brains_stem_cells_eavesdrop_to_find_out_when_to_act
On Using Stem Cells in the Brain to Study Mental Disorders http://www.hopkinsmedicine.org/institute_cell_engineering/_includes/videos/Transcriptions/Song_txn.html
Hopkins Researchers Uncover Key to Antidepressant Response http://www.hopkinsmedicine.org/news/media/releases/hopkins_researchers_uncover_key_to_antidepressant_response
[
| E-mail
Share
]
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
With the death of Google Reader earlier this year, Feedly emerged as one of the major players in RSS, and since its inception it's been using Google's OAuth authentication service to sign in users. With the gradual shift towards Google+ sign-in across Google properties however, Feedly is following suit and introducing G+ sign-in. The transition will take place "this week" on the web and "later this month" through the Feedly Android and iOS apps.
The move to Google+ sign in, Feedly says, allows for easier sign-in on mobile devices and opens up more sharing possibilities, on which it promises to divulge more over the next couple of weeks.
But if Google+ isn't for you, Feedly is also testing Twitter, Facebook and Wordpress login options, with the aim of rolling them out over the next seven weeks.
Source: Feedly Blog
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